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human glioblastoma a172 cell lines  (ATCC)


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    ATCC human glioblastoma a172 cell lines
    Human Glioblastoma A172 Cell Lines, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 2121 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/human+glioblastoma+cells/A-172/bio_rxiv__64898__2026__10__14__759182-37-0-9
    Average 99 stars, based on 2121 article reviews
    human glioblastoma a172 cell lines - by Bioz Stars, 2026-11
    99/100 stars

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    Related Articles

    Polymerase Chain Reaction:

    Article Title: Pro-Synergistic Interaction of Farnesene and Daunorubicin Enhances Apoptotic Vulnerability in U87MG Glioblastoma Cells.
    Article Snippet: Drug synergism, commonly evaluated using Bliss independence and Chou–Talalay models, has therefore become a central concept in GBM treatment design [4–7].. Although preclinical studies have reported synergistic effects between temozolomide and targeted agents, clinical translation remains limited by toxicity, pharmacokinetic constraints, and restricted drug penetration across the blood–brain tumor barrier [8–12].. Anthracyclines remain mechanistically attractive candidates for GBM therapy due to their potent DNA-damaging and reactive oxygen species–generating properties, although their clinical use is limited by systemic toxicity and restricted brain penetration [13, 14].

    Cell Culture:

    Article Title: Phenotypic characterization of two neuroinvasive Toscana virus strains clinically associated with self-limited and persistent infections in human neural cells and brain organoids
    Article Snippet: .. DBTRG.05MG human glioblastoma cells (ATCC CRL-2020) were cultured in RPMI medium (EuroClone) supplemented with 100 U/ml penicillin/streptomycin and 10% FBS at 37 °C in a 5% CO 2 -supplemented incubator. ..

    Article Title: Black maitake induces apoptosis and enhances the efficacy of standard chemotherapy in 2D and 3D U-87 MG glioblastoma cell models.
    Article Snippet: Objectives: Glioblastoma is an aggressive primary brain tumor marked by rapid growth, invasiveness, poor prognosis, and an over 90 % tumor recurrence rate.. Current radiation and chemotherapy treatments are limited by nonselectivity and toxicity, creating a need for safer complementary treatments.. Historically, natural health products (NHPs) have been used medicinally across cultures for their anti-inflammatory and antioxidant effects.

    Article Title: Adaptor protein complex 1 gamma 1 subunit is an important host factor involved in both Zika virus and dengue virus infections
    Article Snippet: .. Human lung carcinoma epithelial cells (A549, ATCC CCL-185), human hepatoma cells (Huh7, provided by Prof. Yiping Li from Sun Yat-sen University), human embryonic kidney cells (HEK 293T, ATCC CRL-3216), and human glioblastoma cells (SNB19, ATCC CRL-2219) were cultured in DMEM supplemented with 10% fetal bovine serum (FBS, Gibco) and 100 U/ml of penicillin–streptomycin (Invitrogen). .. African green monkey kidney cells (Vero, ATCC CCL-81) and baby hamster kidney cells (BHK21, ATCC CCL-10) were maintained in DMEM supplemented with 5% FBS and 100 U/ml of penicillin-streptomycin (Invitrogen).

    In Vitro:

    Article Title: Gold-Doped Hybrid Nanoparticles: A Versatile Tool for Multimodal Imaging of Cell Trafficking
    Article Snippet: .. In vitro toxicity of undoped and gold-doped NPs was evaluated on Human glioblastoma cells (U87-MG, ATCC ® HTB14TM) and human microglia cells (HMC3, ATCC ® CRL3304TM). .. In vitro toxicity of undoped and gold-doped NPs was evaluated on Human glioblastoma cells (U87-MG, ATCC HTB14TM) and human microglia cells (HMC3, ATCC CRL3304TM).



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    Characterization of sEVs and PKH67‐labelled sEVs uptake. The size distribution of sEVs by NTA from the (A) static (collected at D27‐30), (B) orbital shaker (collected at D9–12) and (C) Organoid‐on‐a‐Chip (collected at D27–30) were determined. (D–F) sEVs STEM images from (D) static (at D18–21), (E) orbital shaker (D27–30), (F) Organoid‐on‐a‐Chip (D9–12). (G) The enrichment of sEVs marker Alix and the low abundance of non‐sEV marker Calnexin were detected with Western blotting. (H, I) Uptake of PKH67‐labelled sEVs by <t>U87</t> <t>Glioblastoma</t> cells was demonstrated (scale bar 200 µm). Data are presented as mean ± SEM, * p < 0.05, ** p < 0.01 compared to the control group.
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    Characterization of sEVs and PKH67‐labelled sEVs uptake. The size distribution of sEVs by NTA from the (A) static (collected at D27‐30), (B) orbital shaker (collected at D9–12) and (C) Organoid‐on‐a‐Chip (collected at D27–30) were determined. (D–F) sEVs STEM images from (D) static (at D18–21), (E) orbital shaker (D27–30), (F) Organoid‐on‐a‐Chip (D9–12). (G) The enrichment of sEVs marker Alix and the low abundance of non‐sEV marker Calnexin were detected with Western blotting. (H, I) Uptake of PKH67‐labelled sEVs by <t>U87</t> <t>Glioblastoma</t> cells was demonstrated (scale bar 200 µm). Data are presented as mean ± SEM, * p < 0.05, ** p < 0.01 compared to the control group.
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    Characterization of sEVs and PKH67‐labelled sEVs uptake. The size distribution of sEVs by NTA from the (A) static (collected at D27‐30), (B) orbital shaker (collected at D9–12) and (C) Organoid‐on‐a‐Chip (collected at D27–30) were determined. (D–F) sEVs STEM images from (D) static (at D18–21), (E) orbital shaker (D27–30), (F) Organoid‐on‐a‐Chip (D9–12). (G) The enrichment of sEVs marker Alix and the low abundance of non‐sEV marker Calnexin were detected with Western blotting. (H, I) Uptake of PKH67‐labelled sEVs by <t>U87</t> <t>Glioblastoma</t> cells was demonstrated (scale bar 200 µm). Data are presented as mean ± SEM, * p < 0.05, ** p < 0.01 compared to the control group.
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    ATCC human glioblastoma cells u87 mg
    Characterization of sEVs and PKH67‐labelled sEVs uptake. The size distribution of sEVs by NTA from the (A) static (collected at D27‐30), (B) orbital shaker (collected at D9–12) and (C) Organoid‐on‐a‐Chip (collected at D27–30) were determined. (D–F) sEVs STEM images from (D) static (at D18–21), (E) orbital shaker (D27–30), (F) Organoid‐on‐a‐Chip (D9–12). (G) The enrichment of sEVs marker Alix and the low abundance of non‐sEV marker Calnexin were detected with Western blotting. (H, I) Uptake of PKH67‐labelled sEVs by <t>U87</t> <t>Glioblastoma</t> cells was demonstrated (scale bar 200 µm). Data are presented as mean ± SEM, * p < 0.05, ** p < 0.01 compared to the control group.
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    ATCC human glioblastoma u87 mg cells
    Characterization of sEVs and PKH67‐labelled sEVs uptake. The size distribution of sEVs by NTA from the (A) static (collected at D27‐30), (B) orbital shaker (collected at D9–12) and (C) Organoid‐on‐a‐Chip (collected at D27–30) were determined. (D–F) sEVs STEM images from (D) static (at D18–21), (E) orbital shaker (D27–30), (F) Organoid‐on‐a‐Chip (D9–12). (G) The enrichment of sEVs marker Alix and the low abundance of non‐sEV marker Calnexin were detected with Western blotting. (H, I) Uptake of PKH67‐labelled sEVs by <t>U87</t> <t>Glioblastoma</t> cells was demonstrated (scale bar 200 µm). Data are presented as mean ± SEM, * p < 0.05, ** p < 0.01 compared to the control group.
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    Characterization of sEVs and PKH67‐labelled sEVs uptake. The size distribution of sEVs by NTA from the (A) static (collected at D27‐30), (B) orbital shaker (collected at D9–12) and (C) Organoid‐on‐a‐Chip (collected at D27–30) were determined. (D–F) sEVs STEM images from (D) static (at D18–21), (E) orbital shaker (D27–30), (F) Organoid‐on‐a‐Chip (D9–12). (G) The enrichment of sEVs marker Alix and the low abundance of non‐sEV marker Calnexin were detected with Western blotting. (H, I) Uptake of PKH67‐labelled sEVs by <t>U87</t> <t>Glioblastoma</t> cells was demonstrated (scale bar 200 µm). Data are presented as mean ± SEM, * p < 0.05, ** p < 0.01 compared to the control group.
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    ATCC human glioblastoma cell line u87 mg
    Characterization of sEVs and PKH67‐labelled sEVs uptake. The size distribution of sEVs by NTA from the (A) static (collected at D27‐30), (B) orbital shaker (collected at D9–12) and (C) Organoid‐on‐a‐Chip (collected at D27–30) were determined. (D–F) sEVs STEM images from (D) static (at D18–21), (E) orbital shaker (D27–30), (F) Organoid‐on‐a‐Chip (D9–12). (G) The enrichment of sEVs marker Alix and the low abundance of non‐sEV marker Calnexin were detected with Western blotting. (H, I) Uptake of PKH67‐labelled sEVs by <t>U87</t> <t>Glioblastoma</t> cells was demonstrated (scale bar 200 µm). Data are presented as mean ± SEM, * p < 0.05, ** p < 0.01 compared to the control group.
    Human Glioblastoma Cell Line U87 Mg, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Image Search Results


    Characterization of sEVs and PKH67‐labelled sEVs uptake. The size distribution of sEVs by NTA from the (A) static (collected at D27‐30), (B) orbital shaker (collected at D9–12) and (C) Organoid‐on‐a‐Chip (collected at D27–30) were determined. (D–F) sEVs STEM images from (D) static (at D18–21), (E) orbital shaker (D27–30), (F) Organoid‐on‐a‐Chip (D9–12). (G) The enrichment of sEVs marker Alix and the low abundance of non‐sEV marker Calnexin were detected with Western blotting. (H, I) Uptake of PKH67‐labelled sEVs by U87 Glioblastoma cells was demonstrated (scale bar 200 µm). Data are presented as mean ± SEM, * p < 0.05, ** p < 0.01 compared to the control group.

    Journal: Developmental Neurobiology

    Article Title: Molecular and Proteomic Profiling of Human Cerebral Organoid‑Derived Small Extracellular Vesicles

    doi: 10.1002/dneu.70062

    Figure Lengend Snippet: Characterization of sEVs and PKH67‐labelled sEVs uptake. The size distribution of sEVs by NTA from the (A) static (collected at D27‐30), (B) orbital shaker (collected at D9–12) and (C) Organoid‐on‐a‐Chip (collected at D27–30) were determined. (D–F) sEVs STEM images from (D) static (at D18–21), (E) orbital shaker (D27–30), (F) Organoid‐on‐a‐Chip (D9–12). (G) The enrichment of sEVs marker Alix and the low abundance of non‐sEV marker Calnexin were detected with Western blotting. (H, I) Uptake of PKH67‐labelled sEVs by U87 Glioblastoma cells was demonstrated (scale bar 200 µm). Data are presented as mean ± SEM, * p < 0.05, ** p < 0.01 compared to the control group.

    Article Snippet: Human U87 glioblastoma cells (American Type Culture Collection) were cultured in Dulbecco's modified Eagle medium (DMEM) supplemented with 10% fetal bovine serum (FBS), 1% penicillin/streptomycin.

    Techniques: Marker, Western Blot, Control

    Effects sEVs obtained from static, orbital shaker and Organoid‐on‐a‐Chip conditions on U87 glioblastoma cells. (A) MTT assay for viability of U87 Glioblastoma for static, orbital shaker, and Organoid‐on‐a‐Chip sEVs. (B, C) Representative Ki67/DAPI immunofluorescence images of U87 glioblastoma cells following sEV treatment and quantification of Ki67–DAPI signal overlap using the Manders coefficient. (D, E) Migration of U87 Glioblastoma cells upon sEV treatment was observed by scratch assay up to 48 h. Scale bar 400 µm. (F, G) Diameter of spheres formed on day 2 and day 4 of sEVs treated U87 Glioblastoma by hanging drop sphere formation assay were quantified. Scale bar 200 µm. Data are presented as mean ± SEM, * p < 0.05, ** p < 0.01 compared to the control group.

    Journal: Developmental Neurobiology

    Article Title: Molecular and Proteomic Profiling of Human Cerebral Organoid‑Derived Small Extracellular Vesicles

    doi: 10.1002/dneu.70062

    Figure Lengend Snippet: Effects sEVs obtained from static, orbital shaker and Organoid‐on‐a‐Chip conditions on U87 glioblastoma cells. (A) MTT assay for viability of U87 Glioblastoma for static, orbital shaker, and Organoid‐on‐a‐Chip sEVs. (B, C) Representative Ki67/DAPI immunofluorescence images of U87 glioblastoma cells following sEV treatment and quantification of Ki67–DAPI signal overlap using the Manders coefficient. (D, E) Migration of U87 Glioblastoma cells upon sEV treatment was observed by scratch assay up to 48 h. Scale bar 400 µm. (F, G) Diameter of spheres formed on day 2 and day 4 of sEVs treated U87 Glioblastoma by hanging drop sphere formation assay were quantified. Scale bar 200 µm. Data are presented as mean ± SEM, * p < 0.05, ** p < 0.01 compared to the control group.

    Article Snippet: Human U87 glioblastoma cells (American Type Culture Collection) were cultured in Dulbecco's modified Eagle medium (DMEM) supplemented with 10% fetal bovine serum (FBS), 1% penicillin/streptomycin.

    Techniques: MTT Assay, Immunofluorescence, Migration, Scratch Assay, Tube Formation Assay, Control